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Phase 4 N=83 Randomized Triple-blind Treatment

The Effects of Potassium Citrate on Bone Metabolism

Bone Diseases, Metabolic · Osteoporosis, Postmenopausal

Enrolled (actual)
83
Serious AEs
0.0%
Results posted
Jun 2017
Primary outcome: Primary: Urinary-N-telopeptide — 47.2; 51.6; 44.5; 47 nml BCE/nmol creatinine

Study Design & Population

Study type
Interventional
Phase
Phase 4
Interventions
potassium citrate (Drug)
Age
Adult, Older Adult · 45+ yrs
Sex
Female
Sponsor
Weill Medical College of Cornell University
Primary completion
Apr 2011

Outcome Measures

OutcomeResultp-value
PRIMARY
Urinary-N-telopeptide
47.2; 51.6; 44.5; 47; 50.8; 40.5
PRIMARY
P1NP (Amino-terminal Propeptide of Type I Procollagen)
50.6; 55.5; 48.9; 48.1; 48.7; 43.5
SECONDARY
Number of Participants With Stable Bone Mineral Density (BMD) Over 12 Months at All Sites.
41; 42

Summary

Skeletal buffering of chronic acid loads may contribute to a significant amount of bone loss over time. Evidence from a few small short-term studies suggests that basic compounds, namely potassium citrate and potassium bicarbonate may reduce bone loss and improve bone density. The purpose of this study is to evaluate the effects of potassium citrate on bone metabolism. We hypothesize that administration of potassium citrate to postmenopausal women with osteopenia will reduce bone resorption and improve bone mineral density. Postmenopausal women with osteopenia (T score between -1.0 and -2.5) and no history of fracture will be randomized to either daily potassium citrate or placebo for one year. Primary outcomes will be markers of bone turnover, which will be measured over 12 months. Secondary outcomes will be bone mineral density, compliance, and adverse events.

Eligibility Criteria

Inclusion Criteria

  • Postmenopausal women, more than 2 years post menopause
  • Osteopenia, defined as a T score at the lumbar spine or total hip between -1.0 and -2.5
  • No history of prior fragility fracture

Exclusion criteria

  • Renal insufficiency
  • Use of potassium sparing diuretics
  • Use of potassium supplements
  • Hyperkalemia
  • Secondary causes of osteoporosis or metabolic bone disease
  • Delayed gastric emptying
  • esophageal compression, intestinal obstruction or stricture
  • use of anticholinergic medication
  • active urinary tract infection.
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT00357331). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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