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N/A N=14 Randomized

Pharmacotoxicology of Trichloroethylene Metabolites

Congenital Lactic Acidosis

Enrolled (actual)
14
Serious AEs
7.1%
Results posted
Jun 2013
Primary outcome: Primary: Hypothesize That Subject's Genotype Will Determine How DCA is Metabolized. — 1592; 232 Minutes — p=0.023

Study Design & Population

Study type
Interventional
Phase
N/A
Interventions
Dichloroacetate (DCA) (Drug); Genetic Marker on Chromosome 14q24.3 (Genetic)
Age
Adult · 21+ yrs
Sex
All
Sponsor
University of Florida
Primary completion
Nov 2012

Outcome Measures

OutcomeResultp-value
PRIMARY
Hypothesize That Subject's Genotype Will Determine How DCA is Metabolized.
1592; 232 0.023 sig
SECONDARY
Terminal Half-life (the Amount of Time Needed to Clear One-half of the Dose of Drug)for Environmental Dose 2.5 ug/kg/Day.
65.4; 74.3 0.42

Summary

To establish the relationship between human MAAI haplotype and DCA and tyrosine metabolism. This aim test the postulates that MAAI haplotype determines, and thus can predict,1) dose-dependent DCA kinetics and biotransformation.

Eligibility Criteria

Inclusion Criteria

  • Healthy volunteers

Exclusion Criteria

  • Pregnancy
  • Other medications
  • Psychiatric illness on meds
  • Abnormal labs
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT00874276). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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