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N/A N=18 Randomized Double-blind Basic Science

Study of Dapagliflozin on Mitochondrial Dysfunction and Impaired Insulin Signaling/Action

Insulin Sensitivity · Multiple Mitochondrial Dysfunctions Syndrome

Enrolled (actual)
18
Serious AEs
0.0%
Results posted
Oct 2019
Primary outcome: Primary: Change in Insulin Sensitivity — 3.18; 3.85; 3.57; 5.22 mg/kg.min per µU/ml

Study Design & Population

Study type
Interventional
Phase
N/A
Interventions
Dapagliflozin (Drug); Placebo (Drug)
Age
Adult, Older Adult · 18+ yrs
Sex
All
Sponsor
The University of Texas Health Science Center at San Antonio
Primary completion
Jun 2018

Outcome Measures

OutcomeResultp-value
PRIMARY
Change in Insulin Sensitivity
3.18; 3.85; 3.57; 5.22
SECONDARY
Change in Mitochondrial Function
1.06; 1.3; 1.0; 1.34

Summary

The purpose of this study is to examine the effect of the chronic treatment of type 2 diabetes (T2DM) with dapagliflozin on: (1) mitochondrial gene function/expression and insulin signaling/action and (2) oral glucose tolerance and beta cell function. Dapagliflozin is a potent, highly specific inhibitor of renal glucose transport [SGLT2].

Eligibility Criteria

Inclusion Criteria

  • T2DM
  • Drug Naive Or On Oral Therapy

Exclusion Criteria

  • Insulin Treatment
  • Major Organ Disease
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT01439854). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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