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Phase 3 Completed N=309 Randomized Quadruple-blind Treatment

A Study of the Safety and Efficacy of Two Different Regimens of Mipomersen in Patients With Familial Hypercholesterolemia and Inadequately Controlled Low-Density Lipoprotein Cholesterol

Hypercholesterolemia · Heterozygous Familial
Source: ClinicalTrials.gov NCT01475825 ↗
Enrolled (actual)
309
Serious AEs
20.7%
Results posted
Mar 2019
Primary outcomePrimary: Percent Change From Baseline To Primary Endpoint Visit (PET) In LDL-C In Cohort 1 — -27.17; -6.77; -22.96; -10.62 Percent
◆ Published Evidence
Established
20citations · ~2 / year
Harnessing publicly available genetic data to prioritize lipid modifying therapeutic targets for prevention of coronary heart disease based on dysglycemic risk.
Human genetics · 2016 · Open access · Likely link

Summary

Primary objective: Determine whether mipomersen (ISIS 301012) significantly reduces atherogenic lipid levels in patients with severe heterozygous familial hypercholesterolemia (severe HeFH), defined as low-density lipoprotein cholesterol (LDL-C) levels ≥200 mg/dL plus the presence of coronary heart disease (CHD)/risk equivalents or LDL-C levels ≥300 mg/dL regardless of the presence of CHD/risk equivalents (referred to as Cohort 1) compared to placebo. Two different mipomersen dosing regimens will be studied: subcutaneous (SC) mipomersen 200 mg once weekly versus placebo, and SC mipomersen 70 mg thrice weekly versus placebo. Secondary Objectives: * Determine whether there are qualitative differences between the safety profiles of the 2 dosing regimens and placebo in Cohort 1, patients with HeFH with LDL-C levels ≥160 mg/dL and <200 mg/dL plus the presence of CHD/risk equivalents (referred to as Cohort 2), and the overall study population * Determine whether there are qualitative differences between the tolerability of the 2 dosing regimens and placebo in Cohort 1, Cohort 2, and the overall study population * Further characterize the pharmacokinetics (PK) of the 2 dosing regimens in Cohort 1, Cohort 2, and the overall study population * Determine whether the 2 mipomersen dosing regimens significantly reduce atherogenic lipid levels in Cohort 2 compared to placebo * Obtain additional data regarding ongoing safety and efficacy of mipomersen in patients with FH and inadequately controlled LDL-C who complete the primary efficacy assessment visit (PET) in the Blinded Treatment Period and continue treatment in Open-Label Continuation Period

Linked Publications

  • Harnessing publicly available genetic data to prioritize lipid modifying therapeutic targets for prevention of coronary heart disease based on dysglycemic risk.
    Human genetics · 2016 · 20 citations · Open access · Likely link

Outcome Measures

OutcomeResultp-value
PRIMARY
Percent Change From Baseline To Primary Endpoint Visit (PET) In LDL-C In Cohort 1
-27.17; -6.77; -22.96; -10.62
SECONDARY
Percent Change From Baseline To PET In LDL-C In Cohort 2
-31.20; -9.25; -43.60; -13.57
SECONDARY
Percent Change From Baseline To PET In Apolipoprotein B (Apo B) In Cohort 1
-24.14; -2.83; -21.43; -7.28
SECONDARY
Percent Change From Baseline To PET In Apolipoprotein B (Apo B) In Cohort 2
-30.76; -6.67; -36.34; -3.77
SECONDARY
Percent Change From Baseline To PET in Lipoprotein (a) In Cohort 1
-18.84; -16.85; -27.18; -10.08
SECONDARY
Percent Change From Baseline To PET in Lipoprotein (a) In Cohort 2
-23.41; -11.86; -35.56; 18.79

Eligibility Criteria

Inclusion Criteria

  • Diagnosis of severe hypercholesterolemia (LDL-C ≥300 mg/dL (7.77 mmol/L) or LDL-C ≥200 mg/dL (5.18 mmol/L) with documented coronary heart disease (CHD) or CHD risk equivalents, or diagnosis of Heterozygous Familial Hypercholesterolemia and LDL-C ≥160 mg/dL (4.14 mmol/L) and 6 weeks

Exclusion Criteria

  • Significant health problems in the recent past including heart attack, stroke, coronary syndrome, unstable angina, heart failure, significant arrhythmia, hypertension, blood disorders, liver disease, cancer, digestive disorders, Type I diabetes, or uncontrolled Type II diabetes
  • Apheresis within 3 months prior to Screening or expected to start apheresis during the treatment phase
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT01475825) and the linked publication. Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication. Informational only — not medical advice.

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