Mode
Text Size
Log in / Sign up
N/A N=24 Randomized Prevention

Pilot Trial to Evaluate the Effect of Vitamin D on Melanocyte Biomarkers

Melanoma, Skin

Enrolled (actual)
24
Serious AEs
0.0%
Results posted
Dec 2016
Primary outcome: Primary: Number of Genes That Showed Changes in Expression After Vitamin D Treatment — 270 number of genes

Study Design & Population

Study type
Interventional
Phase
N/A
Interventions
Vitamin D3 (Drug); placebo and vitamin D (Drug)
Age
Adult, Older Adult · 18+ yrs
Sex
Female
Sponsor
Stanford University
Primary completion
May 2014

Outcome Measures

OutcomeResultp-value
PRIMARY
Number of Genes That Showed Changes in Expression After Vitamin D Treatment
270
PRIMARY
Number of Genes Differentialy Regulated in Melanoma That Showed Changes in Expression After Vitamin D Treatment
47
SECONDARY
Vitamin D Toxicity
47.11; 29.83
SECONDARY
Incidence of Hypercalcemia for Vitamin D Toxicity
0; 0

Summary

The purpose of this study is to determine the signaling pathways and changes in gene expression in melanocytes of subjects with a history of non-melanoma skin cancer who are exposed to oral vitamin D. If vitamin D is found to inhibit a signaling pathway involved in the development of melanoma such as BRAF, a protein involved in cell proliferation, then oral vitamin D could be explored further as a chemoprevention for melanoma skin cancer.

Eligibility Criteria

Inclusion Criteria

  • Age 18 - 75
  • Female
  • White race/ethnicity
  • With history of non-melanoma skin cancer
  • Has 12-16 moles upon skin examination
  • Consents to 6-12 moles biopsies over 2-3 clinic visits (2-4 months)
  • Consents to ingesting oral vitamin D3 or placebo daily for 2-4 months
  • Consents to abstaining from other multivitamins during study
  • Consents to research use of their tissue and blood samples
  • Agrees to apply a sunscreen of SPF 45 during study -

Exclusion Criteria

  • History or current evidence of hyperparathyroidism, hypercalcemia, renal calculi, or other renal disease.
  • History or current evidence of malabsorptive illnesses, such as IBD, or liver disease that would impair uptake or metabolism of vitamin D.
  • History or current evidence of hyperthyroidism that would increase metabolism of vitamin D.
  • History or current evidence of immunosuppression (cancer, autoimmune disease) or taking immunosuppressive drugs.
  • Currently taking medications that would affect metabolism of vitamin D (anticonvulsants, corticosteroids, H2-receptor antagonists).
  • Currently taking medications that predispose to hypercalcemia (digoxin, lithium, thiazide diuretics) or other electrolyte disturbances (aluminum hydroxide)
  • Pregnancy
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT01477463). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

Back to search