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N/A N=5

Gene Analysis and Treatment Optimization in Chinese Homozygous Familial Hypercholesterolemia

Homozygous Familial Hypercholesterolemia

Enrolled (actual)
5
Serious AEs
0.0%
Results posted
Feb 2017
Primary outcome: Primary: Number of LDLR Gene Mutations — 7 gene mutations

Study Design & Population

Study type
Observational
Phase
N/A
Interventions
Gene analysis (Genetic); Historical data of lipid-lowering drug administration (Other); Historical data of plasma lipids, xanthoma changes (Other)
Age
Pediatric, Adult, Older Adult
Sex
All
Sponsor
Central South University
Primary completion
Oct 2014

Outcome Measures

OutcomeResultp-value
PRIMARY
Number of LDLR Gene Mutations
7
SECONDARY
LDL-C Reduction Percentage
48.16

Summary

Identify new or novel genes which may impact on cholesterol level, and establish the relationship between those gene mutations with atherosclerosis, as well as responses to lipid-lowering drugs.

Eligibility Criteria

Inclusion criteria

Patients of any age and sex who meet clinical or genetic criteria for hoFH as follows:

  • Cutaneous xanthomata before the age of ten years
  • LDLC > 13 mmol/L before treatment or > 7.76 mmol/L despite treatment
  • Phenotypic features in keeping with HeFH in both parents

Exclusion criteria

Inability of patient, or, if less than 18, a parent, to sign informed consent.

View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT01878604). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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