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Phase 2 N=7 Treatment

A Study to Determine the Effects of NPSP795 on the Calcium-sensing Receptor in Subjects With Autosomal Dominant Hypocalcemia as Measured by PTH Levels and Blood Calcium Concentrations

Autosomal Dominant Hypocalcemia (ADH)

Enrolled (actual)
7
Serious AEs
20.0%
Results posted
Dec 2016
Primary outcome: Primary: Number of Participants With Treatment Emergent Adverse Events (TEAEs) and Treatment Emergent Serious Adverse Events (TESAEs) — 3; 1 participants

Study Design & Population

Study type
Interventional
Phase
Phase 2
Interventions
NPSP795 (Drug)
Age
Adult, Older Adult · 18+ yrs
Sex
All
Sponsor
Shire
Primary completion
May 2015

Outcome Measures

OutcomeResultp-value
PRIMARY
Number of Participants With Treatment Emergent Adverse Events (TEAEs) and Treatment Emergent Serious Adverse Events (TESAEs)
3; 1
PRIMARY
Number of Participants With Clinically Significant Vital Signs and Electrocardiogram (ECG) Abnormalities
PRIMARY
Number of Participants With Potentially Clinically Important Laboratory Abnormalities
1
PRIMARY
Number of Participants With Clinically Significant Abnormalities Related to Physical Examination
1
PRIMARY
Change From Baseline in Ionised Calcium
0.972; 0.950; 0.986; 0.933; 1.025; -0.016
PRIMARY
Change From Baseline in Serum Calcium
1.914; 1.790; 1.852; 1.677; 1.980; -0.114
PRIMARY
Change From Baseline in Urinary Calcium
3.302; 3.912; 2.936; 2.537; 2.065; -0.590
PRIMARY
Change From Baseline in Serum Parathyroid Hormone (PTH)
7.32; 9.14; 9.58; 12.80; 7.40; NA
SECONDARY
Area Under the Plasma Concentration Versus Time Curve (AUC[0-t]) of NPSP795
309.512; 795.766; 1561.746; 1657.057; 2251.818
SECONDARY
Area Under the Concentration Time Curve Extrapolated to Infinity (AUC0-infinity) of NPSP795
2.672; 5.572; 5.437; 7.648; 4.521
SECONDARY
Maximum Observed Drug Concentration (Cmax) of NPSP795 in Plasma
301.900; 745.335; 1467.102; 1515.086; 2141.500
SECONDARY
Elimination Half-life (t1/2) of NPSP795 in Plasma
3.013; 3.913; 4.213; 3.839; 4.000
SECONDARY
Change From Baseline in Fractional Excretion of Calcium (FECa)
0.030; 0.031; 0.027; 0.030; 0.026; 0.002

Summary

This is an open-label study looking at the effects of NPSP795 (a selective calcium receptor antagonist) on activating mutations of the Calcium-sensing receptor in patients with Autosomal Dominant Hypocalcemia. Patients with ADH have low blood calcium levels and an inappropriately increased renal calcium excretion, decreased renal phosphate excretion, and hyperphosphatemia. PTH and blood calcium levels will be tested during and after the IV infusion of NPSP795. Concentrations of NPSP795 and length of time of IV infusion will vary depending on measured levels of ionized calcium.

Eligibility Criteria

Inclusion Criteria

  • Subjects with a heterozygous activating mutation of the CaSR gene (ADH); if not previously confirmed, genetic testing will be performed at the screening visit
  • At least 18 years of age
  • Body mass index (BMI) ≥ 18.5 to 25 ng/mL, the subject will be eligible to continue on to the treatment phase of the study
  • Estimated glomerular filtration rate (GFR) < 25 mL/minute, and/or abnormal hepatic, hematologic, and/or clotting function
  • 12 lead resting electrocardiogram (ECG) with clinically significant abnormalities
  • Concomitant medications with the potential to interfere with NPSP795 metabolism
  • History of thyroid or parathyroid surgery
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT02204579). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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