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N/A N=165

Influence of Genetic Polymorphisms on Ventricular Structure and Function in Patients With Single Ventricle Anatomy

Congenital Heart Disease

Enrolled (actual)
165
Serious AEs
0.0%
Results posted
May 2012
Primary outcome: Primary: To Evaluate the Incidence of the Pre-proendothelin SNP at Nucleotide 5665 — 10 participants

Study Design & Population

Study type
Observational
Phase
N/A
Interventions
Age
Pediatric, Adult, Older Adult
Sex
All
Sponsor
Emory University
Primary completion
Aug 2010

Outcome Measures

OutcomeResultp-value
PRIMARY
To Evaluate the Incidence of the Pre-proendothelin SNP at Nucleotide 5665
10
PRIMARY
Survival
40; 80; 88 0.003 sig

Summary

There are many kids born with congenital heart disease. Some of these defects may lead to the formation of a single ventricle (the heart having only one pumping chamber). These children normally undergo a series of corrective surgeries to help overcome the problems of having just one ventricle. However there are some differences in how well the patients respond to the surgeries. In the adult population, studies have shown that there may be a genetic link that may be responsible for the differences in how patients respond. The investigators would like to study the pediatric population by looking for certain genetic markers in the patients' blood. They will also collect basic health information on each patient.

Eligibility Criteria

Inclusion Criteria

  • Children who currently have functional single ventricle anatomy or have had heart transplantation for single ventricle anatomy who receive their cardiac care at Children's Healthcare of Atlanta, Egleston Hospital.
  • Family agrees to participate in the study following informed consent

Exclusion Criteria

  • Conversion to two ventricle physiology, excluding transplantation.
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT00165984). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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