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Phase 3 N=457 Randomized Quadruple-blind Treatment

Efficacy Study of DiaPep277 in Newly Diagnosed Type 1 Diabetes Patients

Type 1 Diabetes

Enrolled (actual)
457
Serious AEs
8.8%
Results posted
Jun 2016
Primary outcome: Primary: Change From Baseline in Glucagon-stimulated C-peptide AUC at 24 Months — -3.848; -4.348 nmol*minute/L — p=0.2851

Study Design & Population

Study type
Interventional
Phase
Phase 3
Interventions
DiaPep277 (Drug); Placebo (Drug)
Age
Pediatric, Adult · 16+ yrs
Sex
All
Sponsor
Andromeda Biotech Ltd.
Primary completion
Sep 2011

Outcome Measures

OutcomeResultp-value
PRIMARY
Change From Baseline in Glucagon-stimulated C-peptide AUC at 24 Months
-3.848; -4.348 0.2851
SECONDARY
Change From Baseline in Mixed-meal Stimulated C-peptide AUC at 24 Months
-44.33; -43.24 0.7690

Summary

The purpose of this study is to determine if DiaPep277 can effectively protect the internal production of insulin in patients newly diagnosed with type 1 diabetes, by stopping the immune destruction of insulin-producing beta-cells in the pancreas. DiaPep277 acts on the immune system and is expected to prevent further destruction of the beta-cells by stimulating regulatory responses, without causing immunological suppression.

Eligibility Criteria

Inclusion Criteria

  • A diagnosis of type 1 diabetes for up to 3 months at screening
  • Insulin dependency
  • Fasting C-peptide levels >= 0.22 nmol/L
  • Presence of at least 1 of the diabetes-related autoantibodies (IA-2A, GAD or IA)

Exclusion Criteria

  • Pregnancy or intent to conceive in the next 2 years
  • Significant diseases that could affect response to treatment, such as tumors, psychiatric disorders, substance abuse, severe allergies or diabetes-related complications.
  • Patient has immune deficiency or receives immuno-suppressive or cytotoxic drugs.
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT00615264). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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