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N/A N=25 Randomized Supportive Care

Effect of Intraarticular Steroids on Bone Turnover in Osteoarthritis

Osteoarthritis

Enrolled (actual)
25
Serious AEs
0.0%
Results posted
Nov 2016
Primary outcome: Primary: Change in Serum Osteocalcin — -2.18; -0.45; -1.10 ng/mL

Study Design & Population

Study type
Interventional
Phase
N/A
Interventions
Methylprednisolone and Lidocaine (Drug); Placebo and Lidocaine (Drug)
Age
Adult, Older Adult · 40+ yrs
Sex
All
Sponsor
University of Kansas Medical Center
Primary completion
Nov 2009

Outcome Measures

OutcomeResultp-value
PRIMARY
Change in Serum Osteocalcin
-2.18; -0.45; -1.10
PRIMARY
Change in Serum Tartrate-resistant Acid Phosphatase 5b (TRACP-5b)
-0.153; -0.295; -0.148
SECONDARY
Change in Testosterone
0.08
SECONDARY
Change in Serum Cortisol
-1.16; 0.65; 1.30

Summary

Oral and nasal steroids may enhance osteoporosis by suppressing bone formation. Intra-articular steroids may also suppress bone formation, however, the duration or relationship to a steroid dose has not been established. It is hypothesized that intra-articular steroids suppress bone formation transiently, returning to pretreatment levels within four weeks in subjects with osteoarthritis.

Eligibility Criteria

Inclusion Criteria

  • Age > 40 years
  • Male or postmenopausal female
  • Diagnosis of knee osteoarthritis
  • DEXA bone density done within the past 12 months
  • Painful knee, visual analogue scale (VAS) > 4 of (10=worst)

Exclusion Criteria

  • Diabetes Mellitus Type I or II
  • Systemic inflammatory illness
  • Systemic infections which may be aggravated by steroid therapy
  • No current or previous (< 3 years) biphosphate therapy
  • Previous knee replacement surgery
  • No current or previous Parathyroid hormone (PTH) therapy
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT00682357). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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