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N/A N=80 Randomized Quadruple-blind Treatment

Effects of Febuxostat on Adipokines and Kidney Disease in Diabetic Chronic Kidney Disease

Chronic Kidney Disease · Diabetes

Enrolled (actual)
80
Serious AEs
5.0%
Results posted
Oct 2016
Primary outcome: Primary: Change in Thiobarbituric Acid Reactive Substance (TBARS) Concentration in Adipose Tissue From Baseline to 24 Weeks — 34.0; 44.6 percent difference in geometric mean — p=0.84

Study Design & Population

Study type
Interventional
Phase
N/A
Interventions
Febuxostat (Drug); Placebo (Drug)
Age
Adult, Older Adult · 18+ yrs
Sex
All
Sponsor
University of Utah
Primary completion
Oct 2013

Outcome Measures

OutcomeResultp-value
PRIMARY
Change in Thiobarbituric Acid Reactive Substance (TBARS) Concentration in Adipose Tissue From Baseline to 24 Weeks
34.0; 44.6 0.84
PRIMARY
Change in Adiponectin Concentration in Adipose Tissue From Baseline to 24 Weeks
2.3; -4.1 0.73
PRIMARY
Change in Urinary Concentrations of Transforming Growth Factor-beta1 (TGF-beta1) From Baseline to 24 Weeks
29.8; 10.0 0.23
SECONDARY
Change in Tumor Necrosis Factor-α (TNF-α) Concentration in Plasma From Baseline to 24 Weeks
-9.9; -7.4 0.88
SECONDARY
Change in Interleukin-6 (IL-6) Concentration in Plasma From Baseline to 24 Weeks
-24.0; -9.5 0.13
SECONDARY
Change in High Sensitivity C-Reactive Protein (hsCRP) Concentration in Plasma From Baseline to 24 Weeks
12.2; -7.2 0.35

Summary

Hyperuricemia is emerging as a risk factor for development of diabetes and metabolic syndrome. Recently, it was shown in in-vitro cell culture experiments that hyperuricemia induces redox-dependent signaling and oxidative stress in adipocytes. By targeting levels of uric acid with febuxostat it is hypothesized that the levels of oxidative stress in adipose tissue (obtained by fat biopsy) will decrease. Primary aims of the study are to determine whether febuxostat therapy in overweight or obese, diabetic patients with stage 3 Chronic Kidney Disease (CKD) and high serum uric acid levels 1. will affect adipose tissue concentrations of thiobarbituric acid reactive substance (TBARS), a marker of oxidative stress 2. will affect adipose tissue expression and concentrations of adiponectin; and 3. will affect urinary concentrations of transforming growth factor (TGF)- B1.

Eligibility Criteria

Inclusion Criteria

  • Age > 18 years
  • BMI > 25 kg/m2
  • type 2 diabetes
  • serum uric acid ≥ 5.5 mg/dl in men and ≥ 4.6 mg/dl in women
  • eGFR 30-60 mL/min/1.73m2

Exclusion Criteria

  • History of gout
  • concurrent use of azathioprine, mercaptopurine, theophylline, allopurinol, thiazolidinediones or warfarin
  • concurrent use of metformin
  • current antibiotic therapy
  • pregnant women
  • prisoners
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT01350388). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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