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Phase 4 N=85 Treatment

Contribution of Pancreatic αcells Function to Blood Glucose Regulation in Chinese Type 2 Diabetics- the Effect of Sitagliptin on Glucagon Secretion, Insulin Secretion and Insulin Resistance in Chinese Type 2 Diabetics

Insulin Secretion · Insulin Resistance

Enrolled (actual)
85
Serious AEs
0.0%
Results posted
Apr 2017
Primary outcome: Primary: Change From Baseline in Hemoglobin A1c (HbA1c) — -1.08 percentage

Study Design & Population

Study type
Interventional
Phase
Phase 4
Interventions
Sitagliptin (Drug)
Age
Adult · 25+ yrs
Sex
All
Sponsor
Guangwei Li
Primary completion
Aug 2014

Outcome Measures

OutcomeResultp-value
PRIMARY
Change From Baseline in Hemoglobin A1c (HbA1c)
-1.08
SECONDARY
Change From Baseline in Fasting Plasma Glucose (FPG)
-1.99
SECONDARY
Change From Baseline in Postprandial Plasma Glucose (PPG)
-4.69
SECONDARY
Change From Baseline in Insulin Sensitivity
0.64
SECONDARY
Change From Baseline in Insulin Sensitivity in Patients With Different BMI
0.42; 0.72
SECONDARY
Change From Baseline in Pancreatic β Cell Function
2.53
SECONDARY
Change From Baseline in Pancreatic β Cell Function in Patients With Different BMI
2.30; 2.85
SECONDARY
Change From Baseline in Pancreatic α Cell Function
-27.8
SECONDARY
Change From Baseline in Pancreatic α Cell Function in Patients With Different BMI
13.6; -54.0

Summary

1. The purpose of this study is to determine whether Sitagliptin therapy suppress glucagon release and improve glucose control in Chinese type 2 diabetic. 2. There are different effects of Sitagliptin therapy on blood glucose regulation, pancreatic alpha & beta cell function are different in lean (BMI 25) Chinese type 2 diabetics. 3. The purpose of this study is to determine whether glucagon release may contribute over 30% to the hyperglycemia in Chinese type 2 diabetics.

Eligibility Criteria

Inclusion Criteria

  • Age: 25~60 years
  • Duration of disease 7.0mmol/L or/and 2h postprandial blood glucose>11.1mmol/L WHO 1999)
  • Fasting plasma glucose < 10 mmol/L

Exclusion Criteria

  • Type 1 diabetes
  • DKA, infection and other stress status
  • Autoimmune disease
  • Hepatic and renal diseases
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT01610154). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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