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Phase 2 N=91 Treatment

YELLOW II Study: Reduction in Coronary Yellow Plaque, Lipids and Vascular Inflammation by Aggressive Lipid Lowering

Obstructive Coronary Artery Disease · Coronary Artery Disease

Enrolled (actual)
91
Serious AEs
3.5%
Results posted
Feb 2018
Primary outcome: Primary: Correlation Between Plaque Morphology and HDL Functionality — 0.30 beta coefficient

Study Design & Population

Study type
Interventional
Phase
Phase 2
Interventions
rosuvastatin (Drug)
Age
Adult, Older Adult · 18+ yrs
Sex
All
Sponsor
Icahn School of Medicine at Mount Sinai
Primary completion
Apr 2015

Outcome Measures

OutcomeResultp-value
PRIMARY
Correlation Between Plaque Morphology and HDL Functionality
0.30
PRIMARY
Correlation Between the Change in Fibrous Cap Thickness and Hs-CRP
-0.27
SECONDARY
Maximal 4mm Lipid Core Burden Index (LCBI 4mm Max)
416.6; 400.2
SECONDARY
Fibrous Cap Thickness (FCT) by OCT
100.9; 108.6 0.054
SECONDARY
IVUS Imaging Measures
38.75; 38.93; 75.93; 75.79 0.50
SECONDARY
Inflammatory and Lipid Parameters
153.3; 115.0; 86.8; 50.6; 41.2; 42.2
SECONDARY
Lesion LCBI
142.84; 141.93
SECONDARY
LCBI 4mm at Same Anatomical Site
416.6; 383.2
SECONDARY
Change in Atheroma Volume
182.3; 182.7
SECONDARY
Biomarker Release
30.; 0.35
SECONDARY
Correlation of Baseline Lipid Parameters With Baseline LCBI4mm Max
0.07
SECONDARY
Plaque Morphology as Related to Haptoglobin
440.3; 403.0; 411.0; 393.9
SECONDARY
Mechanism of Reverse Cholesterol Transport
0.81; 0.84
SECONDARY
Correlation of Changes in Plaque Morphology
SECONDARY
MACE
33
SECONDARY
MACE
33

Summary

Coronary artery disease (CAD) remains a leading cause of death in most countries. It is well known that the reduction of cholesterol levels by statin therapy is associated with significant decreases in plaque burden. REVERSAL, ASTEROID, and more recently the SATURN II trial showed that in patients with CAD, lipid lowering with atorvastatin or rosuvastatin respectively reduced progression of coronary atherosclerosis, even causing plaque regression of some lesions. CAD clinical events are related to plaque instability due to lipid content and activity within the atherosclerotic plaque. The investigators recently completed the YELLOW I study, and identified that intensive statin therapy (rosuvastatin 40mg) was associated with a reduction in the amount of lipid in obstructive coronary plaques, as measured by near-infrared spectroscopy (NIRS). The YELLOW II study is designed to expand and build upon these results, and to provide mechanistic insights into the potential benefits of intensive statin therapy on atherosclerotic plaques.

Eligibility Criteria

Inclusion Criteria

  • Patients >18 years of age and willing to participate.
  • Fluency in either English or Spanish.
  • Stable patients who will undergo cardiac catheterization and PCI (intent to stent).
  • Patient is willing to go on high-dose cholesterol lowering medication for the duration of the study
  • Signed written Informed Consent.
  • Women of childbearing potential must agree to be on an acceptable method of birth control/contraceptive such as barrier method (condoms/diaphragm); hormonal contraceptives (birth control pills, implants (Norplant) or injections (Depo-Provera)); Intrauterine Device.
  • Proposed non-culprit YELLOW study lesion with max 4mm LCBI ≥ 150.

Exclusion Criteria

  • Patients who have acute myocardial infarction (ST-segment elevation presentation, new Q waves or non-ST segment elevation with CK-MB > 5 times above the upper normal (31.5 ng/ml) within 72 hours).
  • Patients who are in cardiogenic shock.
  • Patients requiring coronary artery bypass graft surgery.
  • Patients with platelet count 2.0 mg/dL.
  • Pregnant women and women of childbearing potential who intend to have children during the duration of the trial.
  • Patients having undergone heart transplantation, or those that may undergo heart transplantation during the study period.
  • Active autoimmune disease.
  • Nursing mothers
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT01837823). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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