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Phase 4 N=23 Randomized Triple-blind Other

The Effect of Dipeptidyl Peptidase 4 Inhibition on Growth Hormone Secretion in Women With Polycystic Ovarian Syndrome

Polycystic Ovary Syndrome

Enrolled (actual)
23
Serious AEs
2.4%
Results posted
May 2020
Primary outcome: Primary: Mean Overnight Growth Hormone Levels — 0.85; 0.84 ng/mL — p=0.918

Study Design & Population

Study type
Interventional
Phase
Phase 4
Interventions
Sitagliptin (Drug); Placebo (Drug)
Age
Adult · 18+ yrs
Sex
Female
Sponsor
Vanderbilt University
Primary completion
Aug 2019

Outcome Measures

OutcomeResultp-value
PRIMARY
Mean Overnight Growth Hormone Levels
0.85; 0.84 0.918
SECONDARY
Early Insulin Secretion During Oral Glucose Tolerance Test
1522.1; 1871.3 0.054
SECONDARY
Area Under the Curve (AUC) for Blood Glucoses During 75 Gram Oral Glucose Tolerance Test
15353.8; 13877.5 0.009 sig
SECONDARY
Visceral Adipose Tissue
1141.9; 1055.1 0.022 sig
SECONDARY
Vascular Function (Endothelium-dependent Vasodilation)
13.63; 13.00 0.943

Summary

Adults with abdominal obesity are at high risk for cardiovascular disease and also exhibit diminished growth hormone (GH) secretion; the latter further contributes to the development of visceral adiposity, impaired fibrinolysis and inflammation.Growth hormone releasing hormone (GHRH), the primary stimulus for endogenous GH secretion, is a substrate of dipeptidyl peptidase 4 (DPP4); inhibition of DPP4 with the currently available anti-diabetic therapy, sitagliptin, may therefore increase GH secretion by decreasing the degradation of GHRH. The proposed research will test the hypothesis that chronic sitagliptin therapy will enhance GH secretion and vascular function while improving glucose tolerance in patients with impaired GH secretion who are at risk for the development of diabetes mellitus and cardiovascular disease, specifically obese women with polycystic ovary syndrome.

Eligibility Criteria

Inclusion Criteria

  • Females, age 18-40 years
  • BMI ≥ 30 kg/m2
  • Diagnosis of polycystic ovary syndrome defined by 2003 Rotterdam criteria as meeting two out of the three below criteria :
  • Oligomenorrhea or amenorrhea
  • clinical or biochemical evidence of hyperandrogenism (hirsutism and/or documented upper normal or elevated serum testosterone in the absence of exogenous hormone therapy or Metformin)
  • documented history of polycystic ovaries on ultrasound examination

Exclusion Criteria

  • Smoking
  • Type 1 or Type 2 Diabetes Mellitus, as defined by a fasting glucose of 126 mg/dL or greater at the time of screening visit or the use of anti-diabetic medication
  • Hypertension, as defined by an untreated seated systolic blood pressure (SBP) greater than 150 mmHg and/or an untreated diastolic blood pressure (DBP) greater than 95 mmHg at the time of screening visit or the use of anti-hypertensive medication
  • History of reported or recorded hypoglycemia (plasma glucose 2 X upper limit of normal range)
  • Treatment with an investigational drug in the 1 month preceding the study
  • Allergy to any of the medications used in this protocol
  • Regular work of a night-shift or unusual schedule which may disrupt circadian rhythm.
  • Personal or Family History (defined as first degree relative) of Pancreatic Cancer
  • Personal history of Pancreatitis or known pancreatic lesions
  • Coagulopathy as defined by history
  • Regular NSAID use, including but not limited to, naproxen, ibuprofen, and aspirin
  • Mental conditions rendering the subject unable to understand the nature, scope, and possible consequences of the study
  • Inability to comply with the protocol, e.g., uncooperative attitude, inability to return for follow-up visits, and unlikelihood of completing the study
  • Any underlying or acute disease requiring regular medication that could possibly pose a threat to the subject or make implementation of the protocol or interpretation of the study results difficult
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT02122380). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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