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Phase 2 N=68 Treatment

Phase 2 Study of MGCD265 in Patients With Non-Small Cell Lung Cancer With Activating Genetic Alterations in MET

Non-Small Cell Lung Cancer

Enrolled (actual)
68
Serious AEs
48.5%
Results posted
Sep 2019
Primary outcome: Primary: Objective Response Rate — 10.7; 15.0; 25.0; 0.0 percentage of participants — p=0.94

Study Design & Population

Study type
Interventional
Phase
Phase 2
Interventions
MGCD265 (Drug)
Age
Adult, Older Adult · 18+ yrs
Sex
All
Sponsor
Mirati Therapeutics Inc.
Primary completion
Apr 2018

Outcome Measures

OutcomeResultp-value
PRIMARY
Objective Response Rate
10.7; 15.0; 25.0; 0.0 0.94
SECONDARY
Duration of Response
85; 170; NA
SECONDARY
Progression Free Survival
3.95; 4.84; 3.39; 2.76
SECONDARY
1-Year Survival Rate
50.47; 34.92; 54.69; 13.89
SECONDARY
Overall Survival
16.32; 7.04; NA; 4.08
SECONDARY
Number of Patients Experiencing Treatment-emergent Adverse Events
28; 20; 8; 12
SECONDARY
Blood Plasma Concentration of MGCD265 - AUC0-6
250.8; 1565; 185.5; 1837
SECONDARY
Blood Plasma Concentration of MGCD265 - Cmax
69.77; 279.2; 60.49; 328.1; 214; 138
SECONDARY
Blood Plasma Concentration of MGCD265 - Ctrough
264; 337; 203; 255; 345
SECONDARY
Blood Plasma Concentration of MGCD265 - Accumulation Ratio AUC0-6
6.42; 9.88
SECONDARY
Blood Plasma Concentration of MGCD265 - Accumulation Ratio Cmax
4.07; 5.35
SECONDARY
Blood Plasma Concentration of MGCD265 - Peak to Trough Ratio
1.09; 1.08
SECONDARY
Blood Plasma Concentration of MGCD265 - Tmax
6.00; 2.00; 6.00; 6.00
SECONDARY
Assess Correlation Between Selected Tumor Gene Alterations Using Different Analytical Techniques in Tumor Tissue and Circulating Tumor Deoxyribonucleic Acid (ctDNA) - MET Activating Mutations
9; 1; 1; 0
SECONDARY
Assess Correlation Between Selected Tumor Gene Alterations Using Different Analytical Techniques in Tumor Tissue and Circulating Tumor Deoxyribonucleic Acid (ctDNA) - MET Gene Amplifications
8; 6; 7; 0
SECONDARY
Assess Change in Genetic Alteration Status in ctDNA With MGCD265 Treatment Over Time in the Selected Population
2; 2; 2; 1
SECONDARY
Blood Plasma Concentration of Soluble MET (sMET) Biomarker
443494.7; 461985.2; 450112.5; 961383.1; 577631.4; 568938.8

Summary

MGCD265 is an orally administered receptor tyrosine kinase inhibitor that targets MET and other receptors. This study is a Phase 2 trial of MGCD265 in patients with locally advanced, unresectable or metastatic non-small cell lung cancer (NSCLC) that has activating genetic changes of the MET gene (mutation or amplification [increase number of gene copies]). Testing for tumor gene changes can be performed in tumor tissue or blood samples. Patients must have previously received treatment with chemotherapy. The number of patients to be enrolled will depend on how many enrolled patients experience tumor size reduction. MGCD265 will be administered orally, twice daily. The study is designed to evaluate whether the number of patients experiencing tumor size reduction is substantially higher than would be expected with other available treatments.

Eligibility Criteria

Inclusion Criteria

  • Diagnosis of non-small cell lung cancer
  • Metastatic or locally advanced disease
  • Prior platinum chemotherapy or immunotherapy
  • Test result showing genetic change in MET tumor gene
  • At least one tumor that can be measured on a radiographic scan

Exclusion Criteria

  • Prior treatment with inhibitor of MET or HGF
  • Prior positive test for EGFR mutation or ALK gene rearrangement
  • Uncontrolled tumor in the brain
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT02544633). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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