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N/A N=10 Randomized Basic Science

Anti-inflammatory Effects of the Fiber

Type 2 Diabetes

Enrolled (actual)
10
Serious AEs
0.0%
Results posted
Oct 2024
Primary outcome: Primary: Peak Change in From Baseline in Reactive Oxygen Species (ROS) Generation — 224; 167 percentage of change

Study Design & Population

Study type
Interventional
Phase
N/A
Interventions
dietary fiber (Dietary_supplement); HFHC meal (Other)
Age
Adult, Older Adult · 18+ yrs
Sex
All
Sponsor
University at Buffalo
Primary completion
Jan 2018

Outcome Measures

OutcomeResultp-value
PRIMARY
Peak Change in From Baseline in Reactive Oxygen Species (ROS) Generation
224; 167
SECONDARY
Peak Change in From Baseline in Nuclear Factor-Kappa B (NFkB) Activation
124; 109
SECONDARY
Percent Change Toll Like Receptor-4 (TLR-4) Levels
181; 135
SECONDARY
Change in Plasma Lipopolysaccharides Level From Baseline.
124; 128

Summary

This study will help elucidate the mechanism underlying the cardioprotective and anti-diabetes effect of dietary fiber by exploring a comprehensive set of inflammatory and oxidative stress markers, based on a contemporary understanding of this process. In addition, there have been very few studies that explored the immediate change in oxidative stress and incretin secretion after fiber intake. In this study, the investigators will be able assess the short term metabolic impact of dietary fiber at great details. The result will contribute to dietary recommendation or designing of fiber supplementation for prevention/treatment of diabetes, obesity and cardiovascular disease.

Eligibility Criteria

Inclusion Criteria

  • Men and women 18 to 80 years of age
  • Non-smoker (last cigarette at least one month ago)
  • Type 2 diabetes for at least 1 year
  • Body mass index > 30 kg/m2

Exclusion Criteria

  • Participation in any other concurrent clinical trials
  • Pregnancy or premenopausal women who are trying to be pregnant
  • Patients who are incompetent to give consent
  • Patients on non-steroidal anti-inflammatory drugs or steroids
  • Concurrent disease that could disrupt intestinal epithelium and increase permeability to endotoxin, ie Celiac and Crohns disease.
  • Hepatic disease (transaminase > 3 times normal)
  • Renal impairment (serum creatinine > 1.5 mg/dl)
  • History of drug or alcohol abuse
  • Use of over the counter or prescribed probiotic supplements.
  • Recent or current antibiotic use.
  • Coronary artery disease (CAD): documented by history of myocardial infarction, angioplasty/stent placement, angina, exercise EKG positive for ischemia or angiographic evidence of CAD
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT02868788). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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