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Phase 2 N=42 Treatment

Safety and Tolerability of RGX-314 (Investigational Product) Gene Therapy for Neovascular AMD Trial

Neovascular Age-related Macular Degeneration · Wet Age-related Macular Degeneration

Enrolled (actual)
42
Serious AEs
28.6%
Results posted
May 2023
Primary outcome: Primary: Safety (Participants With Ocular and Non-ocular AEs (Adverse Events) and SAEs (Serious Adverse Events)) — 6; 6; 6; 12 Participants

Study Design & Population

Study type
Interventional
Phase
Phase 2
Interventions
RGX-314 (Genetic)
Age
Adult, Older Adult · 50+ yrs
Sex
All
Sponsor
REGENXBIO Inc.
Primary completion
Nov 2019

Outcome Measures

OutcomeResultp-value
PRIMARY
Safety (Participants With Ocular and Non-ocular AEs (Adverse Events) and SAEs (Serious Adverse Events))
6; 6; 6; 12; 12
SECONDARY
Safety (Participants With Ocular and Non-ocular AEs and SAEs)
6; 6; 6; 12; 12
SECONDARY
Change From Baseline in BCVA (Best Corrected Visual Acuity)
-7.6; 1.2; 14.0; 0.9; -3.8
SECONDARY
Change From Baseline in CRT (Central Retinal Thickness)
-88.6; 25.3; 2.0; -56.7; -108.2
SECONDARY
Supplemental Injections (Annualized Rate of Supplemental Injections)
10.3; 9.3; 2.8; 4.4; 2.0
SECONDARY
Mean Change From Baseline in Area of CNV (Choroidal Neovascularization)
-0.4; 0.0; -1.2; -1.1; -0.6

Summary

Excessive vascular endothelial growth factor (VEGF) plays a key part in promoting neovascularization and edema in neovascular (wet) age-related macular degeneration (nAMD). VEGF inhibitors (anti-VEGF), including ranibizumab (LUCENTIS®, Genentech) and aflibercept (EYLEA®, Regeneron), have been shown to be safe and effective for treating nAMD and have demonstrated improvement in vision. However, anti-VEGF therapy is administered frequently via intravitreal injection and can be a significant burden to the patients. RGX-314 is a recombinant adeno-associated virus (AAV) gene therapy vector carrying a coding sequence for a soluble anti-VEGF protein. The long-term, stable delivery of this therapeutic protein following a 1 time gene therapy treatment for nAMD could potentially reduce the treatment burden of currently available therapies while maintaining vision with a favorable benefit:risk profile.

Eligibility Criteria

Inclusion Criteria

  • Patients ≥ 50 and ≤ 89 years with a diagnosis of subfoveal CNV (Choroidal neovascularization) secondary to AMD in the study eye receiving prior intravitreal anti-VEGF therapy.
  • BCVA (Best Corrected Visual Acuity) between ≤20/63 and ≥20/400 (≤63 and ≥19 Early Treatment Diabetic Retinopathy Study [ETDRS] letters) for the first patient in each cohort followed by BCVA between ≤20/40 and ≥20/400 (≤73 and ≥19 ETDRS letters) for the rest of the cohort.
  • History of need for and response to anti-VEGF therapy.
  • Response to anti-VEGF at trial entry (assessed by SD-OCT (Spectral Domain Optical Coherence Tomography) at week 1)
  • Must be pseudophakic (status post cataract surgery) in the study eye.
  • AST (Aspartate aminotransferase)/ALT (Alanine aminotransferase) 10 g/dL (males) and > 9 g/dL (females); Platelets > 100 × 10^3/µL; eGFR (Estimated glomerular filtration rate) > 30 mL/min/1.73 m^2
  • Must be willing and able to provide written, signed informed consent.

Exclusion Criteria

  • CNV or macular edema in the study eye secondary to any causes other than AMD.
  • Any condition preventing visual acuity improvement in the study eye, eg, fibrosis, atrophy, or retinal epithelial tear in the center of the fovea.
  • Active or history of retinal detachment in the study eye.
  • Advanced glaucoma in the study eye.
  • History of intravitreal therapy in the study eye, such as intravitreal steroid injection or investigational product, other than anti-VEGF therapy, in the 6 months prior to screening.
  • Presence of an implant in the study eye at screening (excluding intraocular lens).
  • Myocardial infarction, cerebrovascular accident, or transient ischemic attacks within the past 6 months.
  • Uncontrolled hypertension (systolic blood pressure [BP] >180 mmHg, diastolic BP >100 mmHg) despite maximal medical treatment.
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT03066258). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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