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Phase 2 N=60 Randomized Quadruple-blind Treatment

Evolocumab in Acute Coronary Syndrome

Acute Coronary Syndrome

Enrolled (actual)
60
Serious AEs
13.3%
Results posted
Nov 2025
Primary outcome: Primary: Percent Change in LDL-Cholesterol — -68.78; -27.58 percent change

Study Design & Population

Study type
Interventional
Phase
Phase 2
Interventions
Evolocumab (Drug); Placebo (Drug)
Age
Adult, Older Adult · 25+ yrs
Sex
All
Sponsor
Johns Hopkins University
Primary completion
Oct 2024

Outcome Measures

OutcomeResultp-value
PRIMARY
Percent Change in LDL-Cholesterol
-68.78; -27.58
PRIMARY
Change From Baseline in Target to Background Ratio Fluorodeoxyglucose (FDG) Positron Emission Tomography (PET) Scans
-26.7; -10.4
SECONDARY
Left Ventricular Volume as Assessed by Echocardiography
67.80; 59.45; 54.57; 55.63; 62.53; 51.12
SECONDARY
Ejection Fraction as Assessed by Echocardiography
44.28; 49.04; 51.35; 51.90; 50.04; 54.39
SECONDARY
Plasma Proprotein Convertase Subtilisin Kexin-9 (PCSK9) Levels (ng/ml)
244.2; 257.3; NA; 261.9; 215.4; 264.5
SECONDARY
PET-FDG Assessed Vascular Inflammation as Assessed by Standardized Uptake Value (SUV)
3.134705882; 3.708333333 0.2497
SECONDARY
High Sensitivity C-reactive Protein (Hs-CRP) Serum Levels
29.59; 35.48; 15.18; 7.609; 5.174; 3.121
SECONDARY
Change in Serum Levels of Interleukin 6
34.31; 23.08; 8.310; 5.962; 6.941; 2.200
SECONDARY
Serum Levels of Interleukin 10
10.61; 7.948; 9.370; 5.204; 9.291; 5.243

Summary

Vascular and myocardial inflammation are significantly increased in Acute Coronary Syndrome (ACS) patients, are closely correlated to LDL-C levels, and are associated with these adverse consequences in the post-ACS patient population. Serum proprotein convertase subtilisin/kerin type 9 (PCSK9) levels are also increased in ACS, may raise LDL-C, and the investigators' pre-clinical studies indicate that PCSK9 is also a potent inducer of vascular inflammation. The addition of the PCSK9 antibody evolocumab, currently approved to lower LDL-C in certain patient populations, to current medical therapies would appear to be of particular benefit in an important subset of ACS patients, those with non-ST elevation myocardial infarction (NSTEMI) by markedly reducing LDL-C, stabilizing vulnerable plaque, and limiting inflammation-associated myocardial cell loss and resultant dysfunction.

Eligibility Criteria

Inclusion Criteria

  • Non ST segment elevation myocardial infarction
  • Troponin I >/ 5.0 ng/dL
  • Permission of attending physician

Exclusion Criteria

  • ST elevation myocardial infarction
  • Patients requiring invasive hemodynamic support
  • Scheduled for cardiac surgery
  • Current or prior treatment with a PCSK9 antibody
  • Current participation in an intervention clinical trial
  • Female of childbearing potential who has not used acceptable method(s) of birth control for at least one month prior to screening
  • Contraindication to statin therapy
  • Subject likely not able to complete protocol related visits or procedures
  • Latex allergy
  • History of hypersensitivity to any monoclonal antibody
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT03515304). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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