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N/A N=30 Randomized Treatment

Induced and Controlled Dietary Ketosis as a Regulator of Obesity and Metabolic Syndrome Pathologies

Metabolic Syndrome · Diabetes Mellitus, Type 2 · Pre Diabetes

Enrolled (actual)
30
Serious AEs
0.0%
Results posted
Apr 2017
Primary outcome: Primary: Hemoglobin A1c — -.42; -0.37; 0.02 % of hemoglobin — p=0.001

Study Design & Population

Study type
Interventional
Phase
N/A
Interventions
Dietary Ketosis: Regulator of Obesity and Metabolic Syndrome (Behavioral)
Age
Adult, Older Adult · 18+ yrs
Sex
All
Sponsor
Bristlecone Behavioral Health, Inc.
Primary completion
Sep 2016

Outcome Measures

OutcomeResultp-value
PRIMARY
Hemoglobin A1c
-.42; -0.37; 0.02 0.001 sig
SECONDARY
Weight
-28.40; -2.87; -2.43 0.001 sig
SECONDARY
BMI (Body Mass Index)
-4.4; -0.01; -0.42 0.001 sig
SECONDARY
Body Fat Mass (Pounds of Body Fat)
-0.03; -0.01; -0.00 0.001 sig
SECONDARY
Ketones (Blood)
0.24; 0.07; 0.04 0.001 sig

Summary

Original research article entitled Induced and Controlled Dietary Ketosis as a Regulator of Obesity and Metabolic Syndrome by Madeline Gibas for consideration for publication in a clinical journal. This research manuscript builds on previous landmark studies that report that major weight and fat mass loss in type II (T2D) patients who were fed a very low carbohydrate, ketogenic diet. In this manuscript, the investigators outline our research study that showed statistically significant (p < 0.05) changes over time in hemoglobin A1c, weight, BMI, body fat percentage and ketones for patients with metabolic syndrome who were fed a very low carbohydrate diet, ketogenic diet.

Eligibility Criteria

Inclusion Criteria

  • Previously diagnosed with metabolic syndrome

Exclusion Criteria

  • No diagnosis of metabolic syndrome
  • < 18 years old
View full record on ClinicalTrials.gov →

Data sourced from ClinicalTrials.gov (NCT03047447). Outcome figures and adverse-event rates are extracted automatically from the registry's posted results and are provided for clinician reference, not as a substitute for the primary publication.

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